The traditional view of the pathophysiology of stress urinary incontinence (SUI) was that a loss of urethral support was the primary abnormality present.
Recent research has challenged this hypothesis, demonstrating that impaired urethral function plays the key causative role. Improving our understanding of the underlying pathologic mechanisms is important to identifying the cause of treatment failures and for developing novel therapies to treat SUI.
Written by:
Delancey J. Are you the author?
Director of Pelvic Floor Research, Department of Obstetrics and Gynecology, University of Michigan, Ann Arbor, MI.
Reference: Can Urol Assoc J. 2012 Oct;6(5 Suppl 2):S114-5.
doi: 10.5489/cuaj.12195
PubMed Abstract
PMID: 23092768
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